H63D.us: Resources for iron overload and hereditary hemochromatosis

Relief for the most common genetic disorder in the United States


What is Hereditary Hemochromatosis?

Hereditary Hemochromatosis (HH) is a genetic disorder that causes the body to absorb too much iron from food.

Absorb less iron

How is HH diagnosed?

HH is typically diagnosed through blood tests that evaluate the amount of iron in a person's blood.

What are the treatment options?

HH is usually treated through blood draws, also known as phlebotomy, which removes excess iron from the body.

Calculate your treatment

The HFE gene

The HFE gene controls how the body regulates iron. It produces a protein that helps regulate how much iron is absorbed from food. Mutations in the HFE gene, primarily H63D and C282Y, cause a variety of iron issues.

H63D

The H63D mutation in the HFE gene leads to higher levels of iron in the body, which can cause a buildup of iron in organs such as the liver, heart and pancreas. Symptoms may include fatigue, joint pain, abdominal pain and loss of sex drive.

H63D syndrome

H63D syndrome looks a lot like Wilson's disease, but with iron instead of copper.

Easily aggravated by diet, H63D may make for picky eaters or chronically high saturation levels.

Only one in ten people with two H63D errors will show a tendency to load iron in ferritin.

C282Y

The C282Y mutation in the HFE gene is the most common cause of hereditary hemochromatosis, a disorder that causes excessive accumulation of iron in the body. Symptoms may include joint pain, fatigue, abdominal pain, and an enlarged liver.

Classic HH

The extremely high ferritin loading in traditional, two-copy C282Y can be dangerous to the delicate cells of the liver.

The telltale symptom is cirrhosis, or liver-scarring, which can begin to occur over 1000 ferritin. Drinking is a particularly bad combination with this genotype.

H63D/C282Y

H63D/C282Y compound heterozygotes have one copy of each HFE gene mutation. They're less likely to develop severe symptoms than those with two C282Y or H63D mutations. Problems can include joint pain, fatigue, and abdominal pain.

Heterozygosity

The symptoms of the two errors work together fast after a meal, making the compound heterozygote sensitive to iron in food.

Just one copy usually means loading iron 3-5x slower than a double-C282Y. But still loading.

HH-related terms

C282Y
The main error in the HFE gene that promotes iron loading.
H63D
A related error in the HFE gene that causes high saturation and saturation sensitivity.
Ferritin
A protein that stores iron within a cell.
Homozygous
Two copies of an error are present.
Heterozygous
One copy of an error is present (can still have symptoms).
Phlebotomy
A procedure for bloodletting (aka venesection).
Saturation
Transferrin saturation, a measure of how much iron your blood is holding out of how much it can hold.
Serum iron
The iron level within the blood serum (the blood outside the cells).